Overexpression of glutaredoxin-2 reduces myocardial cell death by preventing both apoptosis and necrosis.

Overexpression of glutaredoxin-2 reduces myocardial cell death by preventing both apoptosis and necrosis.

Source :

• Journal of molecular and cellular cardiology [ 1095-8584 ] ; 2008.

Descripteurs français

• KwdFr :
  • Activation enzymatique (MeSH), Animaux (MeSH), Apoptose (MeSH), Cardiolipides (métabolisme), Caspases (métabolisme), Contraction myocardique (MeSH), Cytochromes c (métabolisme), Expression des gènes (MeSH), Femelle (MeSH), Glutarédoxines (génétique), Humains (MeSH), Lésion de reperfusion myocardique (MeSH), Mitochondries du myocarde (enzymologie), Mitochondries du myocarde (métabolisme), Mort cellulaire (MeSH), Myocarde (anatomopathologie), Myocarde (enzymologie), Myocytes cardiaques (anatomopathologie), Myocytes cardiaques (enzymologie), Myocytes cardiaques (métabolisme), Mâle (MeSH), Nécrose (MeSH), Souris (MeSH), Souris transgéniques (MeSH), Stress oxydatif (MeSH), Survie cellulaire (MeSH).
• MESH :
  • anatomopathologie : Myocarde, Myocytes cardiaques.
  • enzymologie : Mitochondries du myocarde, Myocarde, Myocytes cardiaques.
  • génétique : Glutarédoxines.
  • métabolisme : Cardiolipides, Caspases, Cytochromes c, Mitochondries du myocarde, Myocytes cardiaques.
  • Activation enzymatique, Animaux, Apoptose, Contraction myocardique, Expression des gènes, Femelle, Humains, Lésion de reperfusion myocardique, Mort cellulaire, Mâle, Nécrose, Souris, Souris transgéniques, Stress oxydatif, Survie cellulaire.

English descriptors

• KwdEn :
  • Animals (MeSH), Apoptosis (MeSH), Cardiolipins (metabolism), Caspases (metabolism), Cell Death (MeSH), Cell Survival (MeSH), Cytochromes c (metabolism), Enzyme Activation (MeSH), Female (MeSH), Gene Expression (MeSH), Glutaredoxins (genetics), Humans (MeSH), Male (MeSH), Mice (MeSH), Mice, Transgenic (MeSH), Mitochondria, Heart (enzymology), Mitochondria, Heart (metabolism), Myocardial Contraction (MeSH), Myocardial Reperfusion Injury (MeSH), Myocardium (enzymology), Myocardium (pathology), Myocytes, Cardiac (enzymology), Myocytes, Cardiac (metabolism), Myocytes, Cardiac (pathology), Necrosis (MeSH), Oxidative Stress (MeSH).
• MESH :
  • chemical , genetics : Glutaredoxins.
  • chemical , metabolism : Cardiolipins, Caspases, Cytochromes c.
  • enzymology : Mitochondria, Heart, Myocardium, Myocytes, Cardiac.
  • metabolism : Mitochondria, Heart, Myocytes, Cardiac.
  • pathology : Myocardium, Myocytes, Cardiac.
  • Animals, Apoptosis, Cell Death, Cell Survival, Enzyme Activation, Female, Gene Expression, Humans, Male, Mice, Mice, Transgenic, Myocardial Contraction, Myocardial Reperfusion Injury, Necrosis, Oxidative Stress.

Abstract

Affiliations:

• États-Unis
• Connecticut

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